Nonverbal Autism

Discovery Framework

Nonverbality and the Development of NeuroToggle® and BioToggle®

How One Discovery Expanded Into Multiple Frameworks

Kimberly Kitzerow’s work began after her nonverbal autistic daughter could not blow out a candle on her fourth birthday cake. That observation shifted the question from behavior to physiology and eventually led to the development of NeuroToggle® and BioToggle®. This page explains the original discovery, what it changed about the understanding of nonverbal autism, how autism traits and comorbid traits became delineated, and how each framework emerged from that process.

Original Discovery

What Was the Original Discovery?

The Discovery Was About Speech Access

The discovery began when Kitzerow’s nonverbal autistic daughter could not blow out a candle. Blowing out a candle required motor output she could not yet physically access. That shifted the question away from “Why won’t she talk?” and toward “What systems are preventing speech access?”

The key shift was from behavior to mechanism. Nonverbality was no longer treated as one flat autism feature. It became a speech-access problem that needed to be separated, classified, and examined by pathway.
Speech Access

The Systems Required for Speech Production

Speech Requires Language and Motor Circuit Coordination

Speech depends on language circuits and motor circuits developing, functioning, and coordinating continuously. If coordination is disrupted, speech may become physically obstructed even when comprehension, language, and communicative intent are present.

Speech production pathway diagram
Step 1

Speech Mechanisms Delineated

The candle discovery shifted attention toward the speech mechanisms required to physically produce speech.

Step 2

Speech Mechanism Obstruction

If language circuits and motor circuits do not develop, function, or coordinate appropriately, speech output may become physically obstructed.

Step 3

NeuroToggle® Role

NeuroToggle® is a neuroplasticity-informed pedagogy for targeting the neural circuits behind any skill or behavior, including speech, motor planning, sensory regulation, communication, learning, and adaptive development.

Nonverbal Autism

What Did That Change About Nonverbal Autism?

Nonverbality Was Not One Flat Autism Trait

Once speech access was examined physiologically, nonverbality could no longer be treated as one universal autism feature. The same outward presentation could emerge through different mechanisms, meaning nonverbality itself required delineation.

Type 1

Structural or Injury-Based

Speech mechanisms may be impaired by acute injury, early-life trauma, forceps-related damage, nerve disruption, or loss of pathway integrity.

Type 2

Developmental Dysregulation

Speech pathways may develop under altered biological conditions involving nervous system regulation, developmental timing, and pathway formation.

Type 3

Situational Inhibition

Speech access may shut down in selective mutism-type states, overload, emotional intensity, unfamiliar settings, or stress physiology.

The same outward presentation does not necessarily reflect the same mechanism. Separating nonverbality by pathway made it possible to begin separating autism traits from comorbid traits more broadly.
Trait Delineation

Realizing Autism Traits and Comorbid Traits Require Delineation

The Same Presentation Does Not Necessarily Reflect the Same Mechanism

After separating nonverbality from autism itself, Kitzerow began recognizing that many autism-associated traits and comorbidities were being grouped together despite likely involving different underlying mechanisms. Nonverbality, hypotonia, sensory dysregulation, sleep disruption, anxiety, gut dysfunction, immune shifts, motor differences, and regulation changes could not simply be treated as one undifferentiated condition. They required delineation by pathway, mechanism, developmental timing, and physiological involvement.

This delineation process eventually expanded into two separate framework directions. NeuroToggle® focused on targeting skills and behaviors through neural circuit development, while BioToggle® focused on understanding comorbid traits through systems-level regulatory biology.
NeuroToggle®

Using NeuroToggle® to Target Skills and Behaviors

Skills and Behaviors Were Reframed as Neural Circuits

After separating nonverbality from autism traits, Kitzerow applied the same reasoning to skills and behaviors more broadly. NeuroToggle® proposes that cognitive, sensory, motor, communication, academic, and adaptive skills depend on neural circuitry and that those circuits can change through targeted instruction and experience. Speech is one example, not the limit of the framework.

BioToggle®

Understanding Comorbid Traits Through BioToggle®

The Framework Expanded Into Temporally Regulated Systems

As autism traits and comorbid traits became increasingly delineated, Kitzerow began organizing the body into broader categories of regulatory stress-response systems. BioToggle® became the framework for understanding how genetically locked, chronically activated, or situationally activated regulatory systems may shift biological prioritization over time. Within this model, prolonged activation may disrupt temporally regulated development and function across multiple physiological systems.

The Autism and the Comorbidities framework focuses on the delineated outcomes of dysregulated temporally regulated systems. Within this model, autism-associated comorbid traits are examined as the downstream outcomes of prolonged regulatory shifts involving developmental timing, pathway regulation, physiological prioritization, and biological resource allocation over time.
Converging Evidence

Research Began Moving Toward the Same Mechanisms

After the 2020 Discovery and 2022 Dissemination

Following Kitzerow’s initial discovery in 2020 and public dissemination beginning in 2022, increasing amounts of autism research began moving toward motor circuitry, cortical connectivity, neuroplasticity, developmental timing, communication-related pathway regulation, and systems-level physiological mechanisms.

More recent studies are increasingly testing whether targeted neuroplastic interventions involving motor-language systems can influence communication outcomes. This direction overlaps with the same categories of circuitry and pathway regulation Kitzerow publicly argued were involved years earlier.